วันเสาร์ที่ 23 สิงหาคม พ.ศ. 2568

Rheumatoid Arthritis (RA): articular manifestation

Rheumatoid Arthritis (RA): articular manifestation


Introduction

  • RA = chronic, systemic, autoimmune inflammatory arthritis
  • ลักษณะเด่น: symmetrical, peripheral joint inflammation ถ้าไม่ได้ควบคุม erosion, deformity, disability ภายใน 10–20 ปี
  • มีผลต่อ กิจวัตรประจำวันและการทำงาน (เดิน, ลุกนั่ง, เขียน, พิมพ์, ใช้มือ)
  • Extraarticular/systemic features พบได้ใน ~40% ของผู้ป่วย (discussed separately)

Initial Clinical Presentation

Patterns of Onset

  • Classic RA: gradual onset, polyarthritis, morning stiffness, hand/foot/wrist involvement
  • Palindromic rheumatism (~episodic arthritis, hours–days, symptom-free intervals)
    • 30–67% progress to RA
    • RF/ACPA positivity risk progression
    • HCQ ลด risk of progression
  • Monoarthritis: large joint (wrist, knee, shoulder, hip, ankle) อาจพัฒนาเป็น polyarthritis
  • Proximal arthritis: polymyalgia rheumatica (PMR)-like (proximal stiffness) อาจเป็น early RA

Key Symptom

  • Morning stiffness >1 hour hallmark of inflammatory arthritis (nonspecific, but strongly supportive)

Other systemic symptoms

  • Aching, stiffness, fatigue, depression, weight loss
  • Carpal tunnel syndrome อาจเป็นอาการนำ
  • Elderly-onset RA (> 60y): acute polyarthritis + myalgia, fatigue, fever, weight loss

Specific Joint Involvement

Hands

  • Early: pain, swelling, grip strength, squeeze tenderness
  • Signs: dorsal hand swelling (“boxing glove”), palmar tendon thickening/tenosynovitis, nodules tendon rupture
  • Carpal tunnel (1–5% as initial presentation)
  • Deformities (late): MCP subluxation, ulnar drift, swan neck, boutonnière, “bow string sign”

Wrists, Elbows, Shoulders

  • Wrists: early involvement, loss of extension, late = volar subluxation, radial drift, tendon rupture
  • Elbows: effusion, flexion deformity, olecranon bursitis, ulnar neuropathy, subcutaneous nodules
  • Shoulders: late disease painful restriction, frozen shoulder, rotator cuff tear

Feet/Ankles

  • Early: MTP involvement (esp. 5th MTP) tenderness, squeeze test
  • Deformities: lateral drift of toes, cock-up deformities, plantar subluxation callosities
  • Ankle: diffuse swelling (may mimic cellulitis), pain on inversion/eversion
  • Heel pain: retrocalcaneal bursitis, tarsal tunnel syndrome

Knees/Hips

  • Knees: synovial thickening, effusion (patellar tap), restricted ROM, valgus/varus deformity, Baker’s cyst (rupture mimic DVT)
  • Hips: longstanding RA, groin/thigh/back pain, restricted rotation, sometimes trochanteric bursitis

Axial Skeleton

  • Cervical spine (C1–C2 subluxation) risk cord compression, myelopathy, hyperreflexia, Babinski sign
  • Other axial joints: TMJ, SCJ, ACJ, cricoarytenoid (hoarseness/stridor, rare in biologic era)

Laboratory Findings

  • Synovial fluid: inflammatory (WBC 1500–25,000, PMN predominant), low complement, occasionally low glucose
  • Hematology: anemia of chronic disease, thrombocytosis, mild leukocytosis, lymphoma risk, rarely Felty’s syndrome or LGL leukemia
  • Autoantibodies: RF and/or ACPA (+) ใน 75–80%
    • RF: sensitive but less specific
    • ACPA: similar sensitivity, much more specific
    • ANA: 25–30% (clinical significance limited, but SSA+ = risk neonatal lupus/heart block)
  • Acute phase reactants: ESR, CRP usually and correlate with activity

Imaging

  • Plain radiographs (hands/feet):
    • Early: soft tissue swelling, periarticular osteopenia
    • Later: erosions, joint space narrowing, subluxations, deformity
    • By 2 years untreated erosions in ~90%
  • MRI:
    • Detects erosions earlier (7x more than X-ray)
    • Bone marrow edema = predictor of future erosions
    • Detects synovial hypertrophy/proliferation
  • Ultrasound:
    • Sensitive for synovitis, effusion, erosions (esp. MTP)
    • Doppler activity correlates with inflammation

Clinical Course

  • Variable: small vs large joints, number of joints, disease activity fluctuations, remission vs progression
  • Disease activity: fluctuates (weeks–months), DMARDs chance of remission
  • Damage: cumulative, irreversible, linked to inflammation + mechanical stress
  • Remission:
    • Rare without DMARDs
    • Defined by ACR/EULAR criteria (CDAI, SDAI, Boolean 2.0)
    • Radiographic progression can still occur in remission

Key Pearls

  • Classic triad: symmetric polyarthritis + morning stiffness >1 hr + small joint involvement (MCP, PIP, MTP, wrists)
  • Red flags: cervical spine pain, acute neurologic signs suspect subluxation
  • Prognosis: worse with high RF/ACPA titers, early erosions, high inflammatory markers, extraarticular disease
  • Goal: early aggressive DMARDs to induce remission, prevent erosions, preserve function

ไม่มีความคิดเห็น:

แสดงความคิดเห็น