Snoring (Primary snoring / habitual snoring)
ð Definition &
Epidemiology
- āđāļŠีāļĒāļāļāļēāļ vibration āļāļāļ upper airway
soft tissue āļĢāļ°āļŦāļ§่āļēāļ sleep
- āļāļāđāļ้āļ่āļāļĒāļĄāļēāļ
- āļāļēāļĒ ~44%
- āļŦāļิāļ ~28% (āļāļēāļĒุ 30–60 āļี)
- Occasional
snoring āļāļāđāļ้āđāļāļāļุāļāļāļ
ð§ Pathophysiology (Key
clinical concept)
- āļāļĨāđāļāļŦāļĨัāļ:
- ↑ upper airway
resistance
- ↑ pharyngeal
collapsibility
- āļุāļāļŠāļģāļัāļ:
- Anterior
nasal valve = narrowest segment →
highest resistance
- Progression:
1.
mild →
snoring only
2.
moderate →
RERA (respiratory effort related arousal)
3.
severe →
OSA (apnea/hypopnea + desaturation)
⚠️ Associated conditions (āļ้āļāļāļิāļāđāļŠāļĄāļ)
1. Obstructive Sleep Apnea (OSA)
- Snoring
= marker āļāļāļ OSA
- Habitual
snorer:
- āļāļēāļĒ ~34%
- āļŦāļิāļ ~19% →
āļĄี OSA
2. Upper airway narrowing causes
- Obesity
- Nasal
obstruction (allergic rhinitis, septal deviation)
- Craniofacial
abnormality
- Adenotonsillar
hypertrophy
- Hypothyroidism
/ acromegaly
ð Natural history
- Weight
gain →
progression to OSA (strongest factor)
- BMI ↑ 1 unit → AHI ↑ ~5 events/hr
❤️ Clinical consequences
Strong evidence
- ð§
Carotid atherosclerosis (dose-response relationship)
Weak / unclear
- Hypertension
→ likely confounded by
OSA
- Ischemic
heart disease → no
clear independent association
Practical consequence
- Bed
partner sleep disturbance →
significant QoL impact
ð Evaluation (focus =
rule out OSA)
1. History (include bed partner)
- Loud
habitual snoring
- Witnessed
apnea
- Gasping/choking
- Daytime
sleepiness
- Morning
headache, poor concentration
- Risk
factors:
- obesity
- alcohol
- smoking
- sedatives
(benzodiazepine)
2. Physical exam
- Large
neck circumference
- Crowded
oropharynx
- Nasal
obstruction / turbinate hypertrophy
- Craniofacial
abnormality
- Signs
endocrine disease (hypothyroid, acromegaly)
3. Screening tools
- STOP-Bang
- Berlin
questionnaire
4. Indication for sleep study
- Loud
snoring + ≥1:
- witnessed
apnea
- daytime
sleepiness
- obesity
- CV
disease
→ āļŠ่āļ polysomnography / home sleep apnea test
ð§ū Management
ðĒ 1. Conservative
(first-line)
- Weight
loss (strongest evidence)
- Avoid:
- alcohol
(especiallyāļ่āļāļāļāļāļ)
- smoking
- Sleep
position:
- lateral
position (positional therapy)
- Optimize
nasal patency:
- saline
irrigation
- short-term
decongestant
- intranasal
steroid (chronic rhinitis)
ðĄ 2. Devices
Oral appliance (mandibular advancement device)
- First
choice if conservative fail
- Effect:
- ↓ snoring intensity &
frequency (good evidence)
- Side
effects:
- jaw
discomfort, salivation, occlusal change
Nasal dilator
- Low
risk → trial āđāļ้
CPAP
- Effective
āđāļ่:
- compliance
āļ่āļģāļ้āļēāđāļĄ่āļĄี OSA
ðī 3. Surgery (last
option)
- Indication:
refractory cases
- Options:
- UPPP
(uvulopalatopharyngoplasty)
- LAUP
(laser-assisted UPPP)
- Radiofrequency
ablation
- Palatal
implants
⚠️ Limitations:
- Effect
āļĨāļāļĨāļāļĢāļ°āļĒāļ°āļĒāļēāļ§
- āļāļēāļ mask OSA
- complication:
pain, dysphonia, reflux, bleeding
ð§ 4. Myofunctional
therapy
- Oropharyngeal
exercises
- Evidence:
moderate benefit (selected patients)
❌ Not recommended
- Lubricating
nasal sprays →
ineffective
ð Practical clinical
approach (āđāļ้āļāļĢิāļ)
1.
Snoring →
screen OSA first
2.
āļ้āļē OSA → treat OSA
3.
āļ้āļē primary snoring:
o lifestyle
+ positional
o nasal
optimization
o → oral appliance
o → surgery (last)
ðĄ Key take-home for ER /
OPD
- Snoring
≠
benign āđāļŠāļĄāļ → āļ้āļāļāļิāļāļึāļ OSA
- Weight
gain = strongest modifiable risk
- āļ้āļēāļĄี daytime sleepiness + snoring → āļŠ่āļ sleep
study
- Treatment
snoring alone → mainly
QoL indication (partner)
āđāļĄ่āļĄีāļāļ§āļēāļĄāļิāļāđāļŦ็āļ:
āđāļŠāļāļāļāļ§āļēāļĄāļิāļāđāļŦ็āļ