วันอาทิตย์ที่ 22 กุมภาพันธ์ พ.ศ. 2569

Chronic Otitis Media (COM) & Cholesteatoma

Chronic Otitis Media (COM) & Cholesteatoma

1) Definition & Core Concept

Chronic Otitis Media (COM)

  • การติดเชื้อ/อักเสบของ middle ear ± mastoid ร่วมกับ
    👉 Tympanic membrane (TM) perforation แบบเรื้อรัง
  • อาการหลัก:
    otorrhea, hearing loss, aural fullness, บางรายมี otalgia หรือ vertigo

Cholesteatoma

  • ก้อน keratinized squamous epithelium ใน middle ear/mastoid
  • ไม่ใช่ tumor จริง แต่เป็น locally destructive lesion ทำลาย ossicle, bone, intracranial spread ได้

2) Classification (High-yield clinical categories)

2.1 Chronic Otitis Media

Type

ลักษณะ

Inactive (benign COM)

TM perforation + ไม่มี otorrhea

Active COM

TM perforation + มี otorrhea

CSOM (Chronic suppurative OM)

Persistent purulent otorrhea ผ่าน TM perforation

COME

Intact TM + sterile effusion 3 เดือน

WHO: CSOM = otorrhea 2 สัปดาห์ (แต่ ENT มักใช้ 6 สัปดาห์–3 เดือน)


3) Microbiology (CSOM – Clinically important)

Most common pathogens

  • Pseudomonas aeruginosa (สำคัญที่สุด)
  • Staphylococcus aureus (รวม MRSA)

Others

  • Proteus, Klebsiella, E. coli (GNB)
  • Anaerobes (8–59%)
  • Rare:
    • Mycobacterium tuberculosis (ต้องนึกใน TB endemic area เช่น Southeast Asia)
    • Fungi: Aspergillus, Candida

Clinical pearl:

Biofilm formation chronic, refractory infection


4) Pathogenesis (Stepwise mechanism)

4.1 Chronic OM

1.       Recurrent AOM/ET dysfunction

2.       Chronic negative middle ear pressure

3.       TM retraction ± rupture

4.       Chronic perforation persistent infection

5.       Pathogens enter via:

o   Eustachian tube (nasopharynx)

o   External canal (ผ่าน perforated TM)

4.2 Cholesteatoma formation

Primary acquired

  • ET dysfunction TM retraction pocket (pars flaccida)
  • Keratin debris accumulation expanding cholesteatoma

Secondary acquired

  • Squamous epithelium migration ผ่าน TM perforation

Mechanism of bone destruction

  • Collagenase / protease
  • Osteoclastic bone resorption
  • Chronic infection + inflammation

5) Clinical Features (Exam-relevant)

5.1 Chronic Otitis Media

Classic presentation

  • Chronic or recurrent foul-smelling otorrhea
  • Conductive hearing loss
  • Usually painless
  • Worse with water exposure

Physical exam

  • TM perforation + otorrhea
  • Inflamed middle ear mucosa (CSOM)

5.2 Cholesteatoma (Key red-flag entity)

Symptoms

  • Progressive hearing loss
  • Persistent otorrhea
  • Vertigo (late)
  • Facial weakness (advanced)

Otoscopic findings (high yield)

  • Retraction pocket (pars flaccida > pars tensa)
  • Squamous debris / crust
  • “Pearly white mass” behind TM
  • Granulation tissue
  • Attic lesion (posterosuperior quadrant)

Clinical pearl:

Attic retraction + debris = cholesteatoma until proven otherwise


6) Diagnosis & Evaluation (Stepwise clinical approach)

6.1 Clinical diagnosis (Primary care/ER)

Active COM:

  • Chronic otorrhea
  • Perforated TM
  • Minimal systemic symptoms

Inactive COM:

  • Dry TM perforation (incidental finding)

6.2 ENT referral indications (Diagnostic evaluation)

ควร refer ENT ใน:

  • Active COM
  • Suspected cholesteatoma
  • Unclear diagnosis
  • Cranial neuropathy / vertigo / CNS signs (urgent)

6.3 Microbiologic evaluation (CSOM)

  • Obtain culture in ALL CSOM
  • Send:
    • Gram stain
    • Aerobic + anaerobic culture
  • Method:
    Aspiration via otomicroscopy (not ear swab contamination)

Consider special cultures if:

  • TB suspicion (pale TM, multiple perforations, profound HL)
  • Fungal features (itching, refractory)

6.4 Audiometry

Indications:

  • All cholesteatoma
  • Hearing loss
  • Persistent otorrhea

Purpose:

  • Type: conductive vs SNHL
  • Severity baseline
  • Surgical planning

6.5 Imaging (Very high-yield)

CT Temporal Bone (non-contrast)

Indications:

  • ALL cholesteatoma
  • Suspected bone erosion
  • Surgical planning

Findings:

  • Ossicular erosion
  • Scutum erosion
  • Mastoid involvement
  • Fistula formation

MRI (with contrast)

Indications:

  • Suspected intracranial complication
  • Labyrinthine involvement
  • Recurrence monitoring

6.6 Biopsy indications

  • Persistent granulation tissue
  • Pain (atypical for COM)
  • No response after 2–4 weeks treatment
    Rule out malignancy / granulomatous disease

7) Differential Diagnosis (Key distinctions)

  • Otitis externa (painful canal, normal TM)
  • Granulomatosis with polyangiitis
  • Nasopharyngeal carcinoma
  • Langerhans cell histiocytosis
  • TB otitis media

Red flag for malignancy:

  • Persistent otorrhea + pain + treatment failure

8) Management (Clinical Practice Guideline Style)

8.1 General measures (ALL COM)

Water precautions (critical)

  • No swimming/submerging
  • Petroleum jelly cotton while bathing
  • Ear plugs recommended

Aural toilet

  • Suction cleaning by ENT
  • Avoid self-irrigation (risk ototoxicity + worsening infection)

8.2 Inactive COM

  • No specific treatment if asymptomatic
  • Water precautions only
  • Follow-up

8.3 Active COM / CSOM (Key Treatment Strategy)

First-line: Topical antibiotics (preferred)

Recommended agents (safe in TM perforation)

  • Ofloxacin otic
  • Ciprofloxacin ± dexamethasone
    Duration:
  • 2 weeks (extend to 4 weeks if persistent infection)

Rationale:

  • Higher local concentration
  • Better penetration than systemic antibiotics
  • Less systemic resistance

⚠️ Avoid:

  • Aminoglycoside ear drops (ototoxic in perforated TM)

8.4 Systemic antibiotics (Indications)

  • Failure of topical therapy
  • Complicated infection
  • Systemic symptoms (fever, leukocytosis)
  • Invasive infection
  • Resistant organisms

Empiric coverage should include:

  • Pseudomonas
  • MRSA
  • Anaerobes

Example severe regimens:

  • Vancomycin + Cefepime/Ceftazidime + Metronidazole
    (avoid vanc + piperacillin-tazobactam nephrotoxicity)

9) Cholesteatoma Management (Critical Concept)

Definitive treatment = SURGERY

Indication:

  • ALL cholesteatomas (no medical cure)

Goals:

  • Eradicate disease
  • Create dry ear
  • Prevent complications
  • Restore hearing (if possible)

10) Surgical options (ENT)

  • Tympanoplasty (TM repair ± ossicular reconstruction)
  • Tympanomastoidectomy
  • Mastoidectomy (canal wall intact vs canal wall down)
  • Ossicular chain reconstruction

Clinical pearl:

Long process of incus = most vulnerable to erosion


11) Complications (High-yield & life-threatening)

11.1 Extracranial

  • Mastoiditis
  • Facial nerve palsy
  • Labyrinthitis

11.2 Intracranial (0.1–2%)

  • Meningitis
  • Brain abscess (temporal/cerebellar)
  • Lateral sinus thrombosis
  • Intracranial abscess

Red flags:

  • Persistent headache
  • Seizures
  • Fever + otorrhea
  • Focal neurologic deficit
  • Severe ear pain (atypical for COM)

12) Mastoiditis in COM (Emergency pearls)

Symptoms:

  • Postauricular swelling
  • Fever
  • Displaced auricle
  • Mastoid tenderness

Management:

  • Admit
  • IV antibiotics (cover MRSA + Pseudomonas)
  • CT temporal bone
  • ENT surgical consult ± mastoidectomy

13) Key Clinical Pearls (Exam & Practice)

  • Chronic otorrhea + TM perforation = COM until proven otherwise
  • Foul-smelling discharge think CSOM/Pseudomonas
  • Painless chronic discharge = classic COM
  • Pain + treatment failure rule out malignancy
  • Retraction pocket + debris = cholesteatoma
  • Topical fluoroquinolone = first-line CSOM
  • Cholesteatoma = surgical disease (not antibiotic disease)

14) Quick Comparison (AOM vs COM vs Cholesteatoma)

Feature

AOM

COM

Cholesteatoma

Pain

+++

Minimal

Variable

Fever

Possible

Rare

Rare

TM

Bulging

Perforated

Retraction pocket/mass

Otorrhea

Acute

Chronic

Persistent

Treatment

Systemic antibiotics

Topical antibiotics

Surgery


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